Anabolic Steroids: Compound Reference

Long-ester testosterone. Most common injectable AAS. Used for TRT and cycles.

Effects: Elevates testosterone, estradiol (via aromatization), suppresses LH/FSH, increases haemoglobin/haematocrit, worsens lipids (moderate), can elevate liver enzymes slightly

Long-ester testosterone. Similar to enanthate. Common in US TRT protocols.

Effects: Same as Testosterone Enanthate - elevates testosterone, estradiol, suppresses HPTA, increases RBC, worsens lipids

Short-ester testosterone. Faster acting, more frequent injections needed.

Effects: Same effects as other testosterone esters but faster onset and clearance. More stable estradiol levels with EOD injection.

Ultra-long-ester testosterone. Used for TRT with infrequent injections (every 10-14 weeks). Brand names: Nebido, Aveed.

Effects: Same as other testosterone esters: elevates testosterone, estradiol (via aromatization), suppresses LH/FSH, increases haemoglobin/haematocrit, worsens lipids (moderate)

Long-ester 19-nor compound. Popular for mass and joint relief. Deca-Durabolin.

Effects: Suppresses HPTA strongly, can elevate prolactin significantly, generally gentler on lipids than most AAS, beneficial for joints, may remain detectable in blood tests for months after cessation

Short-ester 19-nor compound. NPP - faster acting than Deca.

Effects: Same as Nandrolone Decanoate but faster onset/clearance. Easier to manage prolactin-related sides.

Powerful 19-nor compound. Highly androgenic and anabolic. Short ester.

Effects: Severely worsens lipids (especially HDL), can elevate prolactin, may affect thyroid function, increases haematocrit, can elevate liver enzymes, does not aromatize but has progestogenic activity

Long-ester trenbolone. Same compound, less frequent injections.

Effects: Same as Trenbolone Acetate. Longer ester means sides take longer to resolve if issues arise.

Equipoise (EQ). Very long ester. Known for endurance and lean mass.

Effects: Significant increase in haematocrit/haemoglobin (more than most AAS), moderate lipid impact, can lower estradiol in some individuals (acts as mild AI), long detection time

Anavar. Mild 17α-alkylated DHT derivative. Popular for cutting cycles with relatively low liver stress compared to other orals.

Effects: Significantly lowers SHBG, mild liver stress (17-alpha alkylated but less hepatotoxic than others), worsens lipids (especially HDL suppression), can elevate LDL

Winstrol. 17α-alkylated DHT derivative. Available as oral or water-based injectable.

Effects: Severely impacts lipids (HDL suppression), significant liver stress (oral form), dramatically lowers SHBG, can worsen joint health, elevates liver enzymes

Dianabol. Classic oral mass builder. 17-alpha alkylated.

Effects: Significant liver stress, elevates ALT/AST/GGT, worsens lipids, strong aromatization (high estradiol), water retention, suppresses HPTA

Anadrol. Powerful oral mass builder. 17-alpha alkylated.

Effects: Severe liver stress (highest hepatotoxicity of common orals), dramatically worsens lipids, increases haematocrit significantly, can cause estrogen-like sides despite not aromatizing

DHT derivative. Anti-estrogenic properties. Popular for cutting.

Effects: Can mask elevated estradiol (anti-estrogenic at receptor), moderate lipid impact, may elevate PSA, does not aromatize, moderate HPTA suppression

Mild DHT derivative. Injectable (enanthate) or oral (acetate, 17α-alkylated). Considered one of the safest AAS.

Effects: Mild lipid impact (less than most AAS), minimal liver stress (injectable), does not aromatize, moderate HPTA suppression. Oral form is 17-alpha alkylated with mild liver stress.

Testosterone blend of 4 esters (propionate, phenylpropionate, isocaproate, decanoate). Designed for less frequent injection with multi-phase release.

Effects: Same effects as other testosterone esters — elevates testosterone, estradiol (via aromatization), suppresses LH/FSH, increases haemoglobin/haematocrit, worsens lipids. Mixed esters cause more hormonal fluctuation than single esters.

7-alpha-methyl-19-nortestosterone. Extremely potent 19-nor AAS. Can replace testosterone as a cycle base. Available as acetate ester.

Effects: Very strong HPTA suppression, aromatises to 7-alpha-methyl-estradiol (not detected on standard E2 assays — use sensitive LC-MS/MS), significant lipid impact, may elevate prolactin (19-nor derivative), does not convert to DHT, highly anabolic

Chlorodehydromethyltestosterone (Tbol). Mild oral AAS. 17-alpha alkylated. Originally developed in East Germany for Olympic athletes.

Effects: Moderate liver stress (17-alpha alkylated but milder than Dianabol), worsens lipids (lowers HDL, raises LDL), lowers SHBG, does not aromatise, moderate HPTA suppression

DHT derivative oral AAS. Weak anabolic, strong androgen. Used as an anti-estrogen adjunct and SHBG reducer to increase free testosterone.

Effects: Dramatically lowers SHBG (increases free testosterone), mild anti-estrogenic effect, does not significantly suppress HPTA at low doses, minimal liver stress (not C17-alpha alkylated in the traditional sense), can worsen lipids moderately, may increase DHT-related sides (hair loss, prostate)

Extremely potent oral AAS. 17-alpha alkylated. Used for strength and aggression in powerlifting and pre-contest hardening. Not a mass builder.

Effects: Severe liver stress (one of the most hepatotoxic common orals), significantly elevates ALT/AST/GGT, dramatically worsens lipids, does not aromatise, strong HPTA suppression, can elevate haematocrit, increases aggression

Potent oral AAS. 17-alpha alkylated. Known for rapid dry mass gains. One of the most liver-toxic oral AAS available.

Effects: Severe liver stress (can cause cholestasis), dramatically worsens lipids, significantly elevates ALT/AST, does not aromatise, very strong HPTA suppression, causes lethargy from liver stress at higher doses