5-Amino-1MQ

Small-molecule NNMT inhibitor. Blocks nicotinamide N-methyltransferase to boost NAD+ levels and activate SIRT1. Used for fat loss and metabolic support. Not a peptide but commonly grouped with them.

Overview

Ancillary

Small-molecule NNMT inhibitor. Blocks nicotinamide N-methyltransferase to boost NAD+ levels and activate SIRT1. Used for fat loss and metabolic support. Not a peptide but commonly grouped with them.

Effects on Markers

May improve metabolic markers: preserve NAD+ levels, enhance fat oxidation. May mildly affect lipids (improved profile through metabolic activation). No significant direct impact on hormones, liver enzymes, or haematology in limited data.

Compound Guide

Structure: Synthetic quinolinium compound (not a peptide). Selective inhibitor of NNMT (nicotinamide N-methyltransferase), an enzyme that depletes NAD+ and methyl donors.

Dosage:

  • Standard: 50-100mg/day oral
  • SubQ: 2.5-5mg 1-2x/day (if injectable formulation)
  • Cycle: 8-12 weeks

Administration:

  • Oral capsule (most common) or SubQ injection
  • Take in the morning with or without food
  • Can split dose AM/PM

Key Notes:

  • Mechanism: inhibits NNMT → preserves NAD+ → activates SIRT1 (the "longevity enzyme") → enhanced fat oxidation and metabolic efficiency
  • Not a peptide — small molecule, but commonly sold alongside peptides and stacked with them
  • Preclinical evidence: reduced fat mass while preserving lean muscle in animal models
  • May enhance the effectiveness of exercise for body composition changes
  • NAD+-boosting effects may synergise with anti-aging protocols
  • Very limited human clinical data — most evidence is preclinical
  • Generally well tolerated — mild headache reported occasionally
  • No significant impact on standard blood panels in available data
  • Investigational compound — not approved by any regulatory agency

Usage History

Markers to Monitor

Frequently Asked Questions

Quick Reference

Category

Ancillary

Half-Life

~4-7 hours

Detection Time

N/A

Usage Summary